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Maria João Gameiro de Mascarenhas Saraiva
AuthID:
R-000-BW8
Publications
Confirmed
To Validate
Document Source:
All
Document Type:
All Document Types
Article (244)
Abstract (48)
Review (15)
Book Chapter (12)
Proceedings Paper (10)
Letter (10)
Editorial Material (6)
Note (5)
Correction (3)
Biographical-Item (1)
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Confirmed Publications: 354
31
TITLE:
Preclinical evaluation of RNAi as a treatment for transthyretin-mediated amyloidosis
Full Text
AUTHORS:
Butler, JS; Chan, A; Costelha, S; Fishman, S; Willoughby, JLS; Borland, TD; Milstein, S; Foster, DJ; Goncalves, P; Chen, QM; Qin, J; Bettencourt, BR; Sah, DW; Alvarez, R; Rajeev, KG; Manoharan, M; Fitzgerald, K; Meyers, RE; Nochur, SV;
Saraiva, MJ
;
Zimmermann, TS;
...More
PUBLISHED:
2016
,
SOURCE:
AMYLOID-JOURNAL OF PROTEIN FOLDING DISORDERS,
VOLUME:
23,
ISSUE:
2
INDEXED IN:
Scopus
WOS
CrossRef
:
77
IN MY:
ORCID
32
TITLE:
Transthyretin participates in beta-amyloid transport from the brain to the liver-involvement of the low-density lipoprotein receptor-related protein 1?
Full Text
AUTHORS:
Mobina Alemi; Cristiana Gaiteiro;
Carlos Alexandre Ribeiro
;
Luis Miguel Santos
;
Joao Rodrigues Gomes
;
Sandra Marisa Oliveira
; Pierre Olivier Couraud; Babette Weksler; Ignacio Romero;
Maria Joao Saraiva
;
Isabel Cardoso
;
PUBLISHED:
2016
,
SOURCE:
SCIENTIFIC REPORTS,
VOLUME:
6
INDEXED IN:
Scopus
WOS
CrossRef
:
73
IN MY:
ORCID
33
TITLE:
Transthyretin provides trophic support via megalin by promoting neurite outgrowth and neuroprotection in cerebral ischemia
Full Text
AUTHORS:
Gomes, JR
; Nogueira, RS; Vieira, M;
Santos, SD
; Ferraz Nogueira, JP;
Relvas, JB
;
Saraiva, MJ
;
PUBLISHED:
2016
,
SOURCE:
Cell Death and Differentiation,
VOLUME:
23,
ISSUE:
11
INDEXED IN:
Scopus
CrossRef
:
39
IN MY:
ORCID
34
TITLE:
Polymer-doxycycline conjugates as fibril disrupters: An approach towards the treatment of a rare amyloidotic disease
Full Text
AUTHORS:
Conejos Sanchez, I;
Cardoso, I
; Oteo Vives, M; Romero Sanz, E; Paul, A; Sauri, AR; Morcillo, MA;
Saraiva, MJ
; Vicent, MJ;
PUBLISHED:
2015
,
SOURCE:
JOURNAL OF CONTROLLED RELEASE,
VOLUME:
198
INDEXED IN:
Scopus
WOS
CrossRef
:
27
IN MY:
ORCID
35
TITLE:
Protective Role of Anakinra Against Transthyretin-Mediated Axonal Loss and Cell Death in a Mouse Model of Familial Amyloidotic Polyneuropathy
AUTHORS:
Goncalves, NP
; Teixeira Coelho, M;
Saraiva, MJ
;
PUBLISHED:
2015
,
SOURCE:
JOURNAL OF NEUROPATHOLOGY AND EXPERIMENTAL NEUROLOGY,
VOLUME:
74,
ISSUE:
3
INDEXED IN:
Scopus
WOS
CrossRef
:
6
IN MY:
ORCID
36
TITLE:
Transthyretin Induces Insulin-like Growth Factor I Nuclear Translocation Regulating Its Levels in the Hippocampus
Full Text
AUTHORS:
Vieira, M;
Gomes, JR
;
Saraiva, MJ
;
PUBLISHED:
2015
,
SOURCE:
MOLECULAR NEUROBIOLOGY,
VOLUME:
51,
ISSUE:
3
INDEXED IN:
Scopus
WOS
CrossRef
:
20
IN MY:
ORCID
37
TITLE:
Gene therapy approach to FAP: in vivo influence of T119M in TTR deposition in a transgenic V30M mouse model
Full Text
AUTHORS:
Batista, AR; Gianni, D; Ventosa, M;
Coelho, AV
;
Almeida, MR
;
Sena Esteves, M
;
Saraiva, MJ
;
PUBLISHED:
2014
,
SOURCE:
GENE THERAPY,
VOLUME:
21,
ISSUE:
12
INDEXED IN:
Scopus
WOS
CrossRef
:
8
IN MY:
ORCID
38
TITLE:
Gene therapy approach to FAP: In vivo influence of T119M in TTR deposition in a transgenic V30M mouse model (Gene Therapy advance online publication (2014) 10.1038/gt.2014.86))
AUTHORS:
Batista, AR; Gianni, D; Ventosa, M;
Coelho, AV
;
Almeida, MR
; Sena Esteves, M;
Saraiva, MJ
;
PUBLISHED:
2014
,
SOURCE:
Gene Therapy,
VOLUME:
21,
ISSUE:
12
INDEXED IN:
Scopus
IN MY:
ORCID
39
TITLE:
Glial cells in familial amyloidotic polyneuropathy
AUTHORS:
Nádia Pereira Gonçalves;
Susete Costelha
;
Maria João Saraiva
;
PUBLISHED:
2014
,
SOURCE:
Acta Neuropathologica Communications,
VOLUME:
2,
ISSUE:
1
INDEXED IN:
Scopus
CrossRef
:
11
IN MY:
ORCID
40
TITLE:
Interleukin-1 signaling pathway as a therapeutic target in transthyretin amyloidosis
Full Text
AUTHORS:
Goncalves, NP
; Vieira, P;
Saraiva, MJ
;
PUBLISHED:
2014
,
SOURCE:
AMYLOID-JOURNAL OF PROTEIN FOLDING DISORDERS,
VOLUME:
21,
ISSUE:
3
INDEXED IN:
Scopus
WOS
CrossRef
:
31
IN MY:
ORCID
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