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Isaura Ferreira Tavares
AuthID:
R-000-6F9
Publications
Confirmed
To Validate
Document Source:
All
Document Type:
All Document Types
Article (79)
Abstract (37)
Review (21)
Editorial Material (3)
Proceedings Paper (3)
Correction (3)
Note (2)
Book Chapter (2)
Letter (1)
Unpublished (1)
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Confirmed Publications: 152
111
TITLE:
Neuronal activation at the spinal cord and medullary pain control centers after joint stimulation: A c-fos study in acute and chronic articular inflammation
Full Text
AUTHORS:
Pinto, M
;
Lima, D
;
Tavares, I
;
PUBLISHED:
2007
,
SOURCE:
NEUROSCIENCE,
VOLUME:
147,
ISSUE:
4
INDEXED IN:
Scopus
WOS
CrossRef
:
27
IN MY:
ORCID
|
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CIÊNCIAVITAE
112
TITLE:
Correlation of noxious evoked c-fos expression in areas of the somatosensory system during chronic pain: Involvement of spino-medullary and intra-medullary connections
Full Text
AUTHORS:
Marta Pinto
;
Deolinda Lima
;
Isaura Tavares
;
PUBLISHED:
2006
,
SOURCE:
NEUROSCIENCE LETTERS,
VOLUME:
409,
ISSUE:
2
INDEXED IN:
Scopus
WOS
CrossRef
:
12
IN MY:
ORCID
|
ResearcherID
|
CIÊNCIAVITAE
113
TITLE:
Differential expression of NK1 and GABA(B) receptors in spinal neurones projecting to antinociceptive or pronociceptive medullary centres
Full Text
AUTHORS:
Castro, AR
;
Morgado, C
;
Lima, D
;
Tavares, I
;
PUBLISHED:
2006
,
SOURCE:
BRAIN RESEARCH BULLETIN,
VOLUME:
69,
ISSUE:
3
INDEXED IN:
Scopus
WOS
CrossRef
:
17
IN MY:
ORCID
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ResearcherID
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CIÊNCIAVITAE
114
TITLE:
Inhibition of nociceptive responses of spinal cord neurones during hypertension involves the spinal GABAergic system and a pain modulatory center located at the caudal ventrolateral medulla
Full Text
AUTHORS:
Morato, M
;
Dora Pinho
;
Sousa, T
;
Tavares, I
;
Albino Teixeira, A
;
PUBLISHED:
2006
,
SOURCE:
JOURNAL OF NEUROSCIENCE RESEARCH,
VOLUME:
83,
ISSUE:
4
INDEXED IN:
Scopus
WOS
CrossRef
:
10
IN MY:
ORCID
|
ResearcherID
|
CIÊNCIAVITAE
115
TITLE:
Lesion of the caudal ventrolateral medulla prevents the induction of hypertension by adenosine receptor blockade in rats
Full Text
AUTHORS:
Dora Pinho
;
Sousa, T
;
Morato, M
;
Tavares, I
;
Albino Teixeira, A
;
PUBLISHED:
2006
,
SOURCE:
BRAIN RESEARCH,
VOLUME:
1073,
ISSUE:
1
INDEXED IN:
Scopus
WOS
CrossRef
:
7
IN MY:
ORCID
|
ResearcherID
|
CIÊNCIAVITAE
116
TITLE:
Medullary control of nociceptive transmission: Reciprocal dual communication with the spinal cord
Full Text
AUTHORS:
Almeida, A
;
Leite Almeida, H
;
Tavares, I
;
PUBLISHED:
2006
,
SOURCE:
Drug Discovery Today: Disease Mechanisms,
VOLUME:
3,
ISSUE:
3
INDEXED IN:
Scopus
CrossRef
Handle
IN MY:
ORCID
|
CIÊNCIAVITAE
117
TITLE:
Secondary hyperalgesia in the monoarthritic rat is mediated by GABA(B) and NK1 receptors of spinal dorsal horn neurons: A behavior and c-fos study
Full Text
AUTHORS:
Castro, AR
;
Pinto, M
;
Lima, D
;
Tavares, I
;
PUBLISHED:
2006
,
SOURCE:
NEUROSCIENCE,
VOLUME:
141,
ISSUE:
4
INDEXED IN:
Scopus
WOS
CrossRef
:
22
IN MY:
ORCID
|
ResearcherID
|
CIÊNCIAVITAE
118
TITLE:
The caudal ventrolateral medulla participates in the development and maintenance of adenosine receptor blockade-induced hypertension
AUTHORS:
Dora Pinho
; Marques Lopes, J;
Morato, M
; Sousa, T;
Tavares, I
; Albino Teixeira, A;
PUBLISHED:
2006
,
SOURCE:
16th European Meeting on Hypertension
in
JOURNAL OF HYPERTENSION,
VOLUME:
24
INDEXED IN:
WOS
IN MY:
ORCID
|
ResearcherID
|
CIÊNCIAVITAE
119
TITLE:
The spinal GABAergic system and the VLMlat play a role in the inhibition of spinal dorsal horn neurones during hypertension
AUTHORS:
Morato, M
;
Dora Pinho
;
Sousa, T
; Marques Lopes, J;
Tavares, I
; Albino Teixeira, A;
PUBLISHED:
2006
,
SOURCE:
16th European Meeting on Hypertension
in
JOURNAL OF HYPERTENSION,
VOLUME:
24
INDEXED IN:
WOS
IN MY:
ORCID
|
ResearcherID
|
CIÊNCIAVITAE
120
TITLE:
Imbalance between the expression of NK1 and GABA(B) receptors in nociceptive spinal neurons during secondary hyperalgesia: A c-Fos study in the monoarthritic rat
Full Text
AUTHORS:
Castro, AR
;
Pinto, M
;
Lima, D
;
Tavares, I
;
PUBLISHED:
2005
,
SOURCE:
NEUROSCIENCE,
VOLUME:
132,
ISSUE:
4
INDEXED IN:
Scopus
WOS
CrossRef
:
11
IN MY:
ORCID
|
ResearcherID
|
CIÊNCIAVITAE
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